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Showing 1 to 12 of 139 entries
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Postural Control of Healthy Elderly Individuals Compared to Elderly Individuals with Stroke Sequelae.

The open neurology journal

Alfieri FM, Riberto M, Lopes JA, Filippo TR, Imamura M, Battistella LR.
PMID: 27053967
Open Neurol J. 2016 Mar 15;10:1-8. doi: 10.2174/1874205X01610010001. eCollection 2016.

A stroke and aging process can modify the postural control. We aimed to compare the postural control of health elderly individuals to that of individuals with stroke sequelae. This cross-sectional transversal study was made with individuals capable of walking...

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Alfieri FM, Riberto M, Lopes JA, et al. Postural Control of Healthy Elderly Individuals Compared to Elderly Individuals with Stroke Sequelae. Open Neurol J. 2016;10:1-8doi: 10.2174/1874205X01610010001.
Alfieri, F. M., Riberto, M., Lopes, J. A., Filippo, T. R., Imamura, M., & Battistella, L. R. (2016). Postural Control of Healthy Elderly Individuals Compared to Elderly Individuals with Stroke Sequelae. The open neurology journal, 101-8. https://doi.org/10.2174/1874205X01610010001
Alfieri, Fábio Marcon, et al. "Postural Control of Healthy Elderly Individuals Compared to Elderly Individuals with Stroke Sequelae." The open neurology journal vol. 10 (2016): 1-8. doi: https://doi.org/10.2174/1874205X01610010001
Alfieri FM, Riberto M, Lopes JA, Filippo TR, Imamura M, Battistella LR. Postural Control of Healthy Elderly Individuals Compared to Elderly Individuals with Stroke Sequelae. Open Neurol J. 2016 Mar 15;10:1-8. doi: 10.2174/1874205X01610010001. eCollection 2016. PMID: 27053967; PMCID: PMC4797679.
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Risk Factors for DVT/PE in Patients with Stroke and Intracranial Hemorrhage.

The open neurology journal

Stecker M, Michel K, Antaky K, Cherian S, Koyfmann F.
PMID: 24847389
Open Neurol J. 2014 Apr 04;8:1-6. doi: 10.2174/1874205X01408010001. eCollection 2014.

OBJECTIVE: Deep venous thrombosis (DVT) and pulmonary embolus (PE) are serious problems for patients admitted to the hospital with stroke, subarachnoid hemorrhage (SAH), intracerebral hemorrhage (ICH) and transient ischemic attack (TIA). The purpose of this paper is to further...

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Stecker M, Michel K, Antaky K, et al. Risk Factors for DVT/PE in Patients with Stroke and Intracranial Hemorrhage. Open Neurol J. 2014;8:1-6doi: 10.2174/1874205X01408010001.
Stecker, M., Michel, K., Antaky, K., Cherian, S., & Koyfmann, F. (2014). Risk Factors for DVT/PE in Patients with Stroke and Intracranial Hemorrhage. The open neurology journal, 81-6. https://doi.org/10.2174/1874205X01408010001
Stecker, Mark, et al. "Risk Factors for DVT/PE in Patients with Stroke and Intracranial Hemorrhage." The open neurology journal vol. 8 (2014): 1-6. doi: https://doi.org/10.2174/1874205X01408010001
Stecker M, Michel K, Antaky K, Cherian S, Koyfmann F. Risk Factors for DVT/PE in Patients with Stroke and Intracranial Hemorrhage. Open Neurol J. 2014 Apr 04;8:1-6. doi: 10.2174/1874205X01408010001. eCollection 2014. PMID: 24847389; PMCID: PMC4021206.
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Are Temporal Differences in GDNF and NOS Isoform Induction Contributors to Neurodegeneration? A Fluorescence Microscopy-Based Study.

The open neurology journal

Doursout MF, Liang Y, Schiess MC, Padilla A, Poindexter BJ, Hickson-Bick DL, Bick RJ.
PMID: 27651844
Open Neurol J. 2016 Aug 26;10:67-76. doi: 10.2174/1874205X01610010067. eCollection 2016.

BACKGROUND: Specific factors in Parkinson's disease have become targets as to their protective and degenerative effects. We have demonstrated that cytokines and PD-CSF detrimentally affect microglia and astrocyte growth. While glial cell-derived neurotrophic factor (GDNF) has been recognized as...

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Doursout MF, Liang Y, Schiess MC, et al. Are Temporal Differences in GDNF and NOS Isoform Induction Contributors to Neurodegeneration? A Fluorescence Microscopy-Based Study. Open Neurol J. 2016;10:67-76doi: 10.2174/1874205X01610010067.
Doursout, M. F., Liang, Y., Schiess, M. C., Padilla, A., Poindexter, B. J., Hickson-Bick, D. L., & Bick, R. J. (2016). Are Temporal Differences in GDNF and NOS Isoform Induction Contributors to Neurodegeneration? A Fluorescence Microscopy-Based Study. The open neurology journal, 1067-76. https://doi.org/10.2174/1874205X01610010067
Doursout, Marie-Francoise, et al. "Are Temporal Differences in GDNF and NOS Isoform Induction Contributors to Neurodegeneration? A Fluorescence Microscopy-Based Study." The open neurology journal vol. 10 (2016): 67-76. doi: https://doi.org/10.2174/1874205X01610010067
Doursout MF, Liang Y, Schiess MC, Padilla A, Poindexter BJ, Hickson-Bick DL, Bick RJ. Are Temporal Differences in GDNF and NOS Isoform Induction Contributors to Neurodegeneration? A Fluorescence Microscopy-Based Study. Open Neurol J. 2016 Aug 26;10:67-76. doi: 10.2174/1874205X01610010067. eCollection 2016. PMID: 27651844; PMCID: PMC5009294.
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Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis.

The open neurology journal

Camargo JA, Bertolucci PH.
PMID: 26157521
Open Neurol J. 2015 Jun 10;9:15-20. doi: 10.2174/1874205X01509010015. eCollection 2015.

BACKGROUND: Neurocysticercosis is a parasitic disease that affects the central nervous system. Its main clinical manifestations are epileptic seizures. The objective of this study was to investigate the correlation between neurotransmitter concentrations in cerebrospinal fluid (CSF) and the different...

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Camargo JA, Bertolucci PH. Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis. Open Neurol J. 2015;9:15-20doi: 10.2174/1874205X01509010015.
Camargo, J. A., & Bertolucci, P. H. (2015). Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis. The open neurology journal, 915-20. https://doi.org/10.2174/1874205X01509010015
Camargo, José Augusto, and Bertolucci, Paulo Henrique Ferreira. "Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis." The open neurology journal vol. 9 (2015): 15-20. doi: https://doi.org/10.2174/1874205X01509010015
Camargo JA, Bertolucci PH. Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis. Open Neurol J. 2015 Jun 10;9:15-20. doi: 10.2174/1874205X01509010015. eCollection 2015. PMID: 26157521; PMCID: PMC4485325.
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Dilative Arteriopathy and Leucencephalopathy as Manifestations of a Neurometabolic Disease.

The open neurology journal

Finsterer J, Bastovansky A.
PMID: 26191091
Open Neurol J. 2015 Jun 26;9:28-31. doi: 10.2174/1874205X01509010028. eCollection 2015.

BACKGROUND: Dilative arteriopathy plus leucencephalopathy as a manifestation of a mitochondrial disorder (MID) is rare.CASE REPORT: In a 70yo Caucasian female, height 160cm, weight 62kg, with mild right-sided hemiparesis due to subacute ischemic stroke in the posterior leg of...

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Finsterer J, Bastovansky A. Dilative Arteriopathy and Leucencephalopathy as Manifestations of a Neurometabolic Disease. Open Neurol J. 2015;9:28-31doi: 10.2174/1874205X01509010028.
Finsterer, J., & Bastovansky, A. (2015). Dilative Arteriopathy and Leucencephalopathy as Manifestations of a Neurometabolic Disease. The open neurology journal, 928-31. https://doi.org/10.2174/1874205X01509010028
Finsterer, Josef, and Bastovansky, Adam. "Dilative Arteriopathy and Leucencephalopathy as Manifestations of a Neurometabolic Disease." The open neurology journal vol. 9 (2015): 28-31. doi: https://doi.org/10.2174/1874205X01509010028
Finsterer J, Bastovansky A. Dilative Arteriopathy and Leucencephalopathy as Manifestations of a Neurometabolic Disease. Open Neurol J. 2015 Jun 26;9:28-31. doi: 10.2174/1874205X01509010028. eCollection 2015. PMID: 26191091; PMCID: PMC4503827.
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Solving the Measurement Problem and then Steppin' Out over the Line Riding the Rarest Italian: Crossing the Streams to Retrieve Stable Bioactivity in Majorana Bound States of Dialy zed Human Platelet Lysates.

The open neurology journal

Roedersheimer M.
PMID: 26191092
Open Neurol J. 2015 Jun 26;9:32-44. doi: 10.2174/1874205X01509010032. eCollection 2015.

Exhaustive dialysis (ED) of lysed human platelets against dilute HCl yields stable angiogenic activity. Dialysis against a constrained external volume, with subsequent relaxation of the separation upon opening the dialysis bag, produces material able to maintain phenotypes and viability...

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Roedersheimer M. Solving the Measurement Problem and then Steppin' Out over the Line Riding the Rarest Italian: Crossing the Streams to Retrieve Stable Bioactivity in Majorana Bound States of Dialy zed Human Platelet Lysates. Open Neurol J. 2015;9:32-44doi: 10.2174/1874205X01509010032.
Roedersheimer, M. (2015). Solving the Measurement Problem and then Steppin' Out over the Line Riding the Rarest Italian: Crossing the Streams to Retrieve Stable Bioactivity in Majorana Bound States of Dialy zed Human Platelet Lysates. The open neurology journal, 932-44. https://doi.org/10.2174/1874205X01509010032
Roedersheimer, Mark. "Solving the Measurement Problem and then Steppin' Out over the Line Riding the Rarest Italian: Crossing the Streams to Retrieve Stable Bioactivity in Majorana Bound States of Dialy zed Human Platelet Lysates." The open neurology journal vol. 9 (2015): 32-44. doi: https://doi.org/10.2174/1874205X01509010032
Roedersheimer M. Solving the Measurement Problem and then Steppin' Out over the Line Riding the Rarest Italian: Crossing the Streams to Retrieve Stable Bioactivity in Majorana Bound States of Dialy zed Human Platelet Lysates. Open Neurol J. 2015 Jun 26;9:32-44. doi: 10.2174/1874205X01509010032. eCollection 2015. PMID: 26191092; PMCID: PMC4503829.
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Cerebral Palsy: A Lifelong Challenge Asks for Early Intervention.

The open neurology journal

Panteliadis CP, Hagel C, Karch D, Heinemann K.
PMID: 26191093
Open Neurol J. 2015 Jun 26;9:45-52. doi: 10.2174/1874205X01509010045. eCollection 2015.

One of the oldest and probably well-known examples of cerebral palsy is the mummy of the Pharaoh Siptah about 1196-1190 B.C., and a letter from Hippocrates (460-390 B.C.). Cerebral palsy (CP) is one of the most common congenital or...

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Panteliadis CP, Hagel C, Karch D, et al. Cerebral Palsy: A Lifelong Challenge Asks for Early Intervention. Open Neurol J. 2015;9:45-52doi: 10.2174/1874205X01509010045.
Panteliadis, C. P., Hagel, C., Karch, D., & Heinemann, K. (2015). Cerebral Palsy: A Lifelong Challenge Asks for Early Intervention. The open neurology journal, 945-52. https://doi.org/10.2174/1874205X01509010045
Panteliadis, Christos P, et al. "Cerebral Palsy: A Lifelong Challenge Asks for Early Intervention." The open neurology journal vol. 9 (2015): 45-52. doi: https://doi.org/10.2174/1874205X01509010045
Panteliadis CP, Hagel C, Karch D, Heinemann K. Cerebral Palsy: A Lifelong Challenge Asks for Early Intervention. Open Neurol J. 2015 Jun 26;9:45-52. doi: 10.2174/1874205X01509010045. eCollection 2015. PMID: 26191093; PMCID: PMC4503828.
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Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia.

The open neurology journal

Cogez J, Etard O, Derache N, Defer G.
PMID: 27330574
Open Neurol J. 2016 May 26;10:9-14. doi: 10.2174/1874205X01610010009. eCollection 2016.

OBJECTIVE: The underlying neurophysiologic mechanism responsible for secondary paroxysmal kinesigenic dyskinesia (PKD) is still unclear. Here, we study the pathogenesis of PKD in two patients with a demyelinating lesion in the spinal cord.METHODS: Electromyogram recordings from affected arms of...

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Cogez J, Etard O, Derache N, et al. Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia. Open Neurol J. 2016;10:9-14doi: 10.2174/1874205X01610010009.
Cogez, J., Etard, O., Derache, N., & Defer, G. (2016). Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia. The open neurology journal, 109-14. https://doi.org/10.2174/1874205X01610010009
Cogez, Julien, et al. "Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia." The open neurology journal vol. 10 (2016): 9-14. doi: https://doi.org/10.2174/1874205X01610010009
Cogez J, Etard O, Derache N, Defer G. Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia. Open Neurol J. 2016 May 26;10:9-14. doi: 10.2174/1874205X01610010009. eCollection 2016. PMID: 27330574; PMCID: PMC4891988.
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Study of a Functional Polymorphism in the PER3 Gene and Diurnal Preference in a Colombian Sample.

The open neurology journal

Perea CS, Niño CL, López-León S, Gutiérrez R, Ojeda D, Arboleda H, Camargo A, Adan A, Forero DA.
PMID: 24860629
Open Neurol J. 2014 Apr 18;8:7-10. doi: 10.2174/1874205X01408010007. eCollection 2014.

Polymorphisms in human clock genes have been evaluated as potential factors influencing circadian phenotypes in several populations. There are conflicting results for the association of a VNTR in the PER3 gene and diurnal preference in different studies. The objective...

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Perea CS, Niño CL, López-León S, et al. Study of a Functional Polymorphism in the PER3 Gene and Diurnal Preference in a Colombian Sample. Open Neurol J. 2014;8:7-10doi: 10.2174/1874205X01408010007.
Perea, C. S., Niño, C. L., López-León, S., Gutiérrez, R., Ojeda, D., Arboleda, H., Camargo, A., Adan, A., & Forero, D. A. (2014). Study of a Functional Polymorphism in the PER3 Gene and Diurnal Preference in a Colombian Sample. The open neurology journal, 87-10. https://doi.org/10.2174/1874205X01408010007
Perea, Claudia S, et al. "Study of a Functional Polymorphism in the PER3 Gene and Diurnal Preference in a Colombian Sample." The open neurology journal vol. 8 (2014): 7-10. doi: https://doi.org/10.2174/1874205X01408010007
Perea CS, Niño CL, López-León S, Gutiérrez R, Ojeda D, Arboleda H, Camargo A, Adan A, Forero DA. Study of a Functional Polymorphism in the PER3 Gene and Diurnal Preference in a Colombian Sample. Open Neurol J. 2014 Apr 18;8:7-10. doi: 10.2174/1874205X01408010007. eCollection 2014. PMID: 24860629; PMCID: PMC4031641.
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Investigation of the NOTCH3 and TNFSF7 genes on C19p13 as candidates for migraine.

The open neurology journal

Smith RA, Curtain R, Ovcaric M, Tajouri L, Macmillan J, Griffiths L.
PMID: 19018300
Open Neurol J. 2008;2:1-7. doi: 10.2174/1874205X00802010001. Epub 2008 Apr 23.

To investigate the migraine locus around the C19p13 region through analysis of the NOTCH3 gene (C19p13.2-p13.1), previously shown to be a gene involved in CADASIL and the TNFSF7 gene (C19p13), homologous to the ligands of TNF-alpha and TNF-beta, genes...

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Smith RA, Curtain R, Ovcaric M, et al. Investigation of the NOTCH3 and TNFSF7 genes on C19p13 as candidates for migraine. Open Neurol J. 2008;2:1-7doi: 10.2174/1874205X00802010001.
Smith, R. A., Curtain, R., Ovcaric, M., Tajouri, L., Macmillan, J., & Griffiths, L. (2008). Investigation of the NOTCH3 and TNFSF7 genes on C19p13 as candidates for migraine. The open neurology journal, 21-7. https://doi.org/10.2174/1874205X00802010001
Smith, Robert A, et al. "Investigation of the NOTCH3 and TNFSF7 genes on C19p13 as candidates for migraine." The open neurology journal vol. 2 (2008): 1-7. doi: https://doi.org/10.2174/1874205X00802010001
Smith RA, Curtain R, Ovcaric M, Tajouri L, Macmillan J, Griffiths L. Investigation of the NOTCH3 and TNFSF7 genes on C19p13 as candidates for migraine. Open Neurol J. 2008;2:1-7. doi: 10.2174/1874205X00802010001. Epub 2008 Apr 23. PMID: 19018300; PMCID: PMC2577928.
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Microbial risk factors of cardiovascular and cerebrovascular diseases: potential therapeutical options.

The open neurology journal

Abdalla Abbas M, Guenther A, Galantucci S, Fawi G, Comi G, Kwan J, Corea F.
PMID: 19018303
Open Neurol J. 2008;2:20-4. doi: 10.2174/1874205X00802010020. Epub 2008 May 07.

Infection and inflammation may have a crucial role in the pathogenesis of atherosclerosis. This hypothesis is supported by an increasing number of reports on the interaction between chronic infection, inflammation, and atherogenesis. Assessment of serological and inflammatory markers of...

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Abdalla Abbas M, Guenther A, Galantucci S, et al. Microbial risk factors of cardiovascular and cerebrovascular diseases: potential therapeutical options. Open Neurol J. 2008;2:20-4doi: 10.2174/1874205X00802010020.
Abdalla Abbas, M., Guenther, A., Galantucci, S., Fawi, G., Comi, G., Kwan, J., & Corea, F. (2008). Microbial risk factors of cardiovascular and cerebrovascular diseases: potential therapeutical options. The open neurology journal, 220-4. https://doi.org/10.2174/1874205X00802010020
Abdalla Abbas, Mohammed, et al. "Microbial risk factors of cardiovascular and cerebrovascular diseases: potential therapeutical options." The open neurology journal vol. 2 (2008): 20-4. doi: https://doi.org/10.2174/1874205X00802010020
Abdalla Abbas M, Guenther A, Galantucci S, Fawi G, Comi G, Kwan J, Corea F. Microbial risk factors of cardiovascular and cerebrovascular diseases: potential therapeutical options. Open Neurol J. 2008;2:20-4. doi: 10.2174/1874205X00802010020. Epub 2008 May 07. PMID: 19018303; PMCID: PMC2577933.
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Spinal Manipulation is Not an Emerging Risk Factor for Stroke Nor is it Major Head/Neck Trauma. Don't Just Read the Abstract!.

The open neurology journal

Smith DL, Cramer GD.
PMID: 21687558
Open Neurol J. 2011;5:46-7. doi: 10.2174/1874205X01105010046. Epub 2011 May 30.

No abstract available.

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Smith DL, Cramer GD. Spinal Manipulation is Not an Emerging Risk Factor for Stroke Nor is it Major Head/Neck Trauma. Don't Just Read the Abstract!. Open Neurol J. 2011;5:46-7doi: 10.2174/1874205X01105010046.
Smith, D. L., & Cramer, G. D. (2011). Spinal Manipulation is Not an Emerging Risk Factor for Stroke Nor is it Major Head/Neck Trauma. Don't Just Read the Abstract!. The open neurology journal, 546-7. https://doi.org/10.2174/1874205X01105010046
Smith, Dean L, and Cramer, Gregory D. "Spinal Manipulation is Not an Emerging Risk Factor for Stroke Nor is it Major Head/Neck Trauma. Don't Just Read the Abstract!." The open neurology journal vol. 5 (2011): 46-7. doi: https://doi.org/10.2174/1874205X01105010046
Smith DL, Cramer GD. Spinal Manipulation is Not an Emerging Risk Factor for Stroke Nor is it Major Head/Neck Trauma. Don't Just Read the Abstract!. Open Neurol J. 2011;5:46-7. doi: 10.2174/1874205X01105010046. Epub 2011 May 30. PMID: 21687558; PMCID: PMC3115591.
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Showing 1 to 12 of 139 entries